Speaker articles

Take a look at what some of our Metabolites: Organellar communication in health and disease speakers have published recently and chat with them about their work in Sitges, Spain.

Keynote speakers

Nav Chandel, Northwestern University, USA

Mitochondrial respiration in microglia is essential for response to demyelinating injury but not proliferation

Mitochondrial integrated stress response controls lung epithelial cell fate

Heather Christofk, University of California, Los Angeles, USA

Atlas of fetal metabolism during mid-to-late gestation and diabetic pregnancy

ZFP36-mediated mRNA decay regulates metabolism

Speakers

Monther Abu-Remaileh, Stanford University, USA

Glycerophosphodiesters inhibit lysosomal phospholipid catabolism in Batten disease

The Batten disease gene product CLN5 is the lysosomal bis(monoacylglycero)phosphate synthase

Kivanc Birsoy, The Rockefeller University, USA

Glycosphingolipid synthesis mediates immune evasion in KRAS-driven cancer

Integrative genetic analysis identifies FLVCR1 as a plasma-membrane choline transporter in mammals

Donita Brady, University of Pennsylvania, USA

BRAFV600E-Driven Lung Adenocarcinoma Requires Copper to Sustain Autophagic Signaling and Processing

The copper chaperone CCS facilitates copper binding to MEK1/2 to promote kinase activation

Elvan Boke, Centre for Genomic Regulation (CRG), Spain

Mouse oocytes sequester aggregated proteins in degradative super-organelles

Oocytes maintain ROS-free mitochondrial metabolism by suppressing complex I

Lulu Cambronne, University of Texas, USA

SLC25A51 decouples the mitochondrial NAD+/NADH ratio to control proliferation of AML cells

Dynamics of SLC25A51 reveal preference for oxidized NAD+ and substrate led transport

Mark Febbraio, Monash University, Australia

Therapeutic blockade of ER stress and inflammation prevents NASH and progression to HCC

Diet-induced gut dysbiosis and inflammation: Key drivers of obesity-driven NASH

Pekka Katajisto, University Helsinki, Finland & Karolinska Institute, Sweden

Cellular shape reinforces niche to stem cell signaling in the small intestine

Metabolic determination of cell fate through selective inheritance of mitochondria

Judith Klumperman, University Medical Center Utrecht, USA

An optimized protocol for immuno-electron microscopy of endogenous LC3

Quantitative correlative microscopy reveals the ultrastructural distribution of endogenous endosomal proteins

Natalie Krahmer, Institute for Diabetes and Obesity, Helmholtz Diabetes Center, Germany

A spatiotemporal proteomic map of human adipogenesis

EPAC1 enhances brown fat growth and beige adipogenesis

James Olzmann, Berkeley, University of California, USA

Parallel CRISPR-Cas9 screens identify mechanisms of PLIN2 and lipid droplet regulation

Identification of structurally diverse FSP1 inhibitors that sensitize cancer cells to ferroptosis

Rushika Perera, University of California San Francisco, USA

Coordinated Transcriptional and Catabolic Programs Support Iron-Dependent Adaptation to RAS-MAPK Pathway Inhibition in Pancreatic Cancer

Autophagy in cancer cell remodeling and quality control

Bao-Liang Song, Wuhan University, China

A sterol analog inhibits hedgehog pathway by blocking cholesterylation of smoothened

Inhibition of ASGR1 decreases lipid levels by promoting cholesterol excretion

Jessica Spinelli, UMass Chan Medical School, USA

Oxygen-Independent Assays to Measure Mitochondrial Function in Mammals

Selenium reduction of ubiquinone via SQOR suppresses ferroptosis

Phillip White, Duke Department of Medicine, USA

BCAA-nitrogen flux in brown fat controls metabolic health independent of thermogenesis

Muscle-Liver Trafficking of BCAA-Derived Nitrogen Underlies Obesity-Related Glycine Depletion

Roberto Zoncu, Berkeley Research, University of California, USA
Lysosomal GPCR-like protein LYCHOS signals cholesterol sufficiency to mTORC1

NPC1-mTORC1 Signaling Couples Cholesterol Sensing to Organelle Homeostasis and Is a Targetable Pathway in Niemann-Pick Type C

Antonio Zorzano, IRB Barcelona - Institute for Research in Biomedicine, Spain

TP53INP2-dependent activation of muscle autophagy ameliorates sarcopenia and promotes healthy aging

Disruption of mitochondrial dynamics triggers muscle inflammation through interorganellar contacts and mitochondrial DNA mislocation

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